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Newest in this topic

  1. Astroglial expression of human alpha(1)-antichymotrypsin enhances alzheimer-like pathology in amyloid protein precursor transgenic mice 2000-12-01
  2. Transforming growth factor-beta 1 induces neuronal and astrocyte genes: tubulin alpha 1, glial fibrillary acidic protein and clusterin 1994-02-01
  3. Astrocytes in Alzheimer's disease gray matter express alpha 1-antichymotrypsin mRNA 1989-11-01

Studies

3 studies carry this tag.

Date range

1989 to 2000

Top journals

  • The American journal of pathology 2
  • Neuroscience 1

Newest first, then by address. Each study has a usefulness score out of 100 (rubric version 4.0); tap a score to see what makes it up. Tags were assigned automatically from each study's title and summary. The same studies as a filter in the library.

  1. Astroglial expression of human alpha(1)-antichymotrypsin enhances alzheimer-like pathology in amyloid protein precursor transgenic mice

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The American journal of pathology 2000-12-01 Biomarkers

    Proteases and their inhibitors play key roles in physiological and pathological processes.

  2. Transforming growth factor-beta 1 induces neuronal and astrocyte genes: tubulin alpha 1, glial fibrillary acidic protein and clusterin

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. Neuroscience 1994-02-01 Biomarkers

    Transforming growth factor-beta 1 was studied as a possible regulator of messenger RNAs in astrocytes and neurons that increase after hippocampal deafferentation by perforant path transection: tubulin alpha 1, clusterin and glial fibrillary acidic protein messenger RNA.

  3. Astrocytes in Alzheimer's disease gray matter express alpha 1-antichymotrypsin mRNA

    ✓ Peer-reviewed alzhub usefulness score 36 out of 100. The American journal of pathology 1989-11-01 Biomarkers

    The serine protease inhibitor alpha 1-antichymotrypsin (ACT) has been shown to be tightly associated with the amyloid found in plaque cores and blood vessels in the brains of patients with Alzheimer's disease (AD).

All topic tags with their study counts