News
What's happening in Alzheimer's care, research funding, and treatment — summarized simply, each item linked to the original reporting.
Free to read, “May need a subscription” and “Archived copy”: what they mean
Whether an article is free to read is a guess, not a test of your access. Each item is one of three:
- No label: free to read (314 of 362). The publisher’s page marks it free, or the outlet publishes free to read: Medical Xpress, ScienceDaily, NIH (incl. NIA and PMC), Alzheimer's Association, Alzheimer's News Today, NPR, Alzheimer's Disease International, or a .gov site.
- May need a subscription (13 of 362). The publisher’s own page marks the article as not free to read, or — when the page says nothing — the outlet sells subscriptions: The New York Times, STAT, Scientific American, The Washington Post, The Wall Street Journal, Financial Times, The Economist, Bloomberg, The Atlantic, The New Yorker, Wired, Los Angeles Times, The Boston Globe, The Times, The Telegraph, New Scientist, Endpoints News, Science. Each badge says which.
- “Free to read? Not known” (35 of 362). The outlet is on neither list and its page said nothing. We do not call these paywalled.
Archived copy links go to a copy the Internet Archive (web.archive.org) saved, dated the day it was saved. We show one only where the Internet Archive reported a working saved copy when we checked; an archived copy of a subscription article sometimes shows only its opening. 267 of 362 items have one.
Rapid tau spread may explain why midlife Alzheimer's progresses more rapidly
Patients with early-onset Alzheimer's disease, whose symptoms start before age 65, have more extensive deposits of tau, a hallmark protein of the condition, compared with patients whose symptoms start at 65-plus, a new study found. …
Read at Medical Xpress →Hidden ancestral links found in motor neuron disease and dementia could speed up gene discovery
Researchers at the Macquarie University Brain Institute and Clinic have helped accelerate the search for disease-causing genes by building new genetic 'families.' …
Read at Medical Xpress →Scientists discover new Alzheimer’s drug that repairs DNA damage and reduces brain inflammation
Scientists have discovered that a drug originally developed for spinal cord injuries can tackle several key features of Alzheimer’s disease, including DNA damage and inflammation. …
Read at ScienceDaily →New human data hints at psychedelics' potential to combat inflammation
Lab studies have shown that psychedelics like psilocybin can lower inflammation and may be protective for a range of illnesses. Now, researchers are publishing some of the first data in humans that signals this holds potential for patients with neurodegenerative diseases like…
Read at NPR → Archived copy (23 Oct 2026), Internet ArchiveMouse study reveals molecular link between high-fat diets and impaired brain function
The molecular mechanisms linking a high-calorie diet to cognitive impairment remain unclear. Researchers at the Institute of Science Tokyo, Japan, investigated this association in mice…
Read at Medical Xpress →Scientists find a way to fight Alzheimer’s brain damage without removing toxic tau
Scientists have found a promising way to protect the brain from Alzheimer’s-related damage by blocking immune cells that contribute to the destruction of neurons. …
Read at ScienceDaily → Archived copy (20 Oct 2026), Internet Archive"Mind-blowing" Alzheimer’s breakthrough: Two hours of sleep restored without clearing brain plaques
Scientists have uncovered an unexpected culprit behind sleep loss in Alzheimer’s disease: overactive immune cells in the brain. By temporarily removing these cells, researchers restored more than two hours of sleep per day in mice, revealing a promising new target for treatment.
Read at ScienceDaily → Archived copy (19 Oct 2026), Internet ArchiveLab-designed molecule reduces amyloid beta, the protein linked to Alzheimer's disease, in mice
A research team led by the Molecular and Cellular Neurobiotechnology group at the Institute for Bioengineering of Catalonia (IBEC), in collaboration with Míriam Royo's laboratory at the Institute for Advanced Chemistry of Catalonia (IQAC-CSIC)…
Read at Medical Xpress →Brain cells meant to protect us may be fueling cognitive decline
Scientists have uncovered a surprising cause of age-related cognitive decline involving brain cells previously thought to be entirely beneficial. …
Read at ScienceDaily → Archived copy (17 Oct 2026), Internet ArchiveScientists find a youth protein that helps aging brain cells act young again
Scientists have discovered that a protein linked to youth helps protect the brain’s immune cells from age-related decline. Restoring the protein in older mice improved the cells’ ability to clear debris and reduced signs of inflammation…
Read at ScienceDaily → Archived copy (18 Oct 2026), Internet ArchiveResearchers restore neuron function in mice with Alzheimer's disease
Researchers were able to restore the function of neurons linked to cognitive function and information processing in mice that mimic key features of Alzheimer's disease, showing early promise for the development of a similar targeted therapy in humans. …
Read at Medical Xpress →Who gets Alzheimer's drugs? Race, vulnerability and health status shape access
The first disease-modifying drugs for Alzheimer's disease offer patients the ability to slow cognitive decline by targeting amyloid plaques in the brain. But a new study suggests that not all patients have the same opportunity to receive these treatments.
Rare study reveals a striking divide inside an Alzheimer’s patient’s brain
A rare examination of an Alzheimer’s patient’s brain revealed that regions cleared of amyloid plaques had remarkably little of the harmful tau buildup associated with memory loss, while neighboring areas showed extensive damage. …
Molecular glue degraders show early promise in reducing Alzheimer's-linked brain plaques
A research team led by Indiana University School of Medicine scientists has discovered a new method for reducing the formation of amyloid plaques…
Read at Medical Xpress → Archived copy (15 Oct 2026), Internet ArchiveDementia Cases Rising; Tylenol's Effect on Brain Organoids; Early-Onset Alzheimer's
(MedPage Today) -- An estimated 11.4 million new dementia cases occurred worldwide in 2023, and the number is projected to jump to 24.7 million new cases a year by 2050, a meta-analysis of 22 cohort studies showed. (JAMA Neurology) Investigational...
Free to read? Not known for this outlet.
Read at MedPage Today → Archived copy (15 Oct 2026), Internet ArchiveStudy offers new clues about how genetic risk for Alzheimer's affects brain immune cells
Researchers at the University of Colorado Anschutz and Pacific Northwest National Laboratory have found a new clue that may help explain why some people are more likely to develop Alzheimer's disease. …
Read at Medical Xpress → Archived copy (13 Oct 2026), Internet ArchiveDrug combination that reduces oxygen levels in tissues improves Parkinson's symptoms in mice
In a new study, Broad Institute researchers report that two small-molecule chemicals that reduce oxygen delivery to cells improved symptoms of neurodegeneration in a mouse model of Parkinson's disease. …
Read at Medical Xpress →Scientists find hidden driver of aging and a simple amino acid that fights memory loss in mice
Scientists discovered that declining levels of a protein called Menin may help drive aging, and restoring it in older mice reversed several age-related changes in just 30 days. …
Read at ScienceDaily → Archived copy (12 Oct 2026), Internet ArchiveBrain tissue removed during surgery reveals potential way to curb inflammation linked to Alzheimer's
Inflammatory conditions in the brain, including traumatic brain injury and degenerative diseases like Alzheimer's, could be targeted with an existing drug thanks to new research that shows it can work in brain cells.
Why it matters: New research in brain cells reveals an existing drug could potentially curb inflammatory conditions linked to Alzheimer's and traumatic brain injury.
Read at Medical Xpress → Archived copy (8 Oct 2026), Internet ArchiveHuman tau seeds trigger disease-specific shapes in mouse brains, new study finds
Tau is a protein that occurs naturally in the brain. It is essential for proper neuron development and for how neurons communicate with each other. The same protein, however, when misshapen or misfolded, can act as a driver of neurodegenerative diseases. …
Why it matters: Misshapen tau protein can act as a driver of neurodegenerative diseases despite its natural role in neuron development and communication.
Read at Medical Xpress → Archived copy (5 Oct 2026), Internet ArchiveResearchers slow neurodegeneration in mice by barring T cells from the brain
A new study in mice led by Washington University in St. Louis researchers suggests that stopping certain immune cells from entering the brain could limit neurodegeneration.
Why it matters: Stopping certain immune cells from entering the brain could limit neurodegeneration, according to a study in mice led by Washington University in St. Louis researchers.
May need a subscription Based on the outlet (STAT), not a check of this article.
Read at STAT News → Archived copy (30 Sep 2026), Internet ArchiveAlcohol and the Brain; The Next Alzheimer's Frontier; Mice With Human Brain Cells
(MedPage Today) -- The New York Times looked at what alcohol does to the brain and what amount, if any, may be safe. Ongoing phase I/II studies of ifezuntirgene inilparvovec (AMT-130) in Huntington's disease showed continued slowing of disease...
Free to read? Not known for this outlet.
Read at MedPage Today → Archived copy (1 Oct 2026), Internet ArchiveScientists restore a brain protein and reverse signs of aging in mice
Scientists found that declining levels of the brain protein Menin may help drive aging, with lower levels in mice linked to inflammation, cognitive decline, weaker bones, and thinner skin. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveA major Alzheimer’s risk gene may shrink brain cells years before symptoms
Researchers have uncovered a possible reason why the Alzheimer’s risk gene APOE4 can disrupt the brain long before memory problems begin. In mice, APOE4 increased a protein called Nell2, shrinking neurons and making memory circuits unusually active. …
Read at ScienceDaily → Archived copy (1 Oct 2026), Internet ArchiveScientists discover a hidden brain shift that begins around age 50
Beginning around age 50, the brain’s memory center appears to lose many of its longtime immune cells and replace them with more inflammatory ones. This surprising shift may help explain how normal aging sets the stage for Alzheimer’s disease and dementia.
Read at ScienceDaily → Archived copy (27 Sep 2026), Internet ArchiveA hidden Alzheimer’s tipping point may decide who gets dementia
Scientists have identified a possible tipping point that helps determine whether Alzheimer’s-related brain changes lead to dementia. The key may lie in how the brain’s immune cells respond to plaques and tau…
APOE2 may protect the brain from Alzheimer’s and aging
The longevity-linked APOE2 gene appears to protect brain cells by reducing DNA damage and helping neurons recover from stress. The finding could open a new path toward treatments that mimic APOE2’s defenses in people at higher genetic risk for Alzheimer’s.
Alzheimer’s breakthrough: Scientists restore two hours of sleep without clearing brain plaques
Researchers have uncovered a surprising culprit behind sleep loss in Alzheimer’s disease: the brain’s own immune cells. In mice with amyloid plaques, overactive microglia triggered inflammation that kept the brain from getting enough deep, restorative sleep. …
Read at ScienceDaily → Archived copy (22 Sep 2026), Internet ArchiveScientists discover a protein that protects the brain from Alzheimer’s damage
A protein called SORLA may act as a powerful defense against the toxic tau tangles involved in Alzheimer’s disease. Mice with extra SORLA had less tau accumulation, less brain atrophy…
Read at ScienceDaily → Archived copy (2 Oct 2026), Internet ArchiveNew Alzheimer's drug repairs DNA damage and reduces brain inflammation
A drug originally developed for spinal cord injury may offer a fresh approach to treating Alzheimer’s disease. In mouse studies, KCL-286 repaired dangerous DNA damage, reduced inflammation…
Read at ScienceDaily → Archived copy (17 Jul 2026), Internet ArchiveFollow new items in a feed reader: Atom feed.