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% Source: https://alzhub.org/research?category=genetics_trem2&author=Wang+M
% 5 entries, in the order the page lists them. Built from alzhub.org's own records.
% 0 entries have no readable author in the source record and carry no author field.

@article{li2022-35658903,
  author  = {Li, R. Y. and Qin, Q. and Yang, H. C. and Wang, Y. Y. and Mi, Y. X. and Yin, Y. S. and Wang, M. and Yu, C. J. and others},
  title   = {{TREM2 in the pathogenesis of AD: a lipid metabolism regulator and potential metabolic therapeutic target}},
  journal = {Molecular neurodegeneration},
  year    = {2022},
  doi     = {10.1186/s13024-022-00542-y},
  pmid    = {35658903},
  url     = {https://doi.org/10.1186/s13024-022-00542-y}
}

@article{zhou2022-35359989,
  author  = {Zhou, L. and Wang, M. and Guo, H. and Hou, J. and Zhang, Y. and Li, M. and Wu, X. and Chen, X. and others},
  title   = {{Integrated Analysis Highlights the Immunosuppressive Role of TREM2+ Macrophages in Hepatocellular Carcinoma}},
  journal = {Frontiers in immunology},
  year    = {2022},
  doi     = {10.3389/fimmu.2022.848367},
  pmid    = {35359989},
  url     = {https://doi.org/10.3389/fimmu.2022.848367}
}

@article{hauremirande2019-30283032,
  author  = {Haure-Mirande, J. V. and Wang, M. and Audrain, M. and Fanutza, T. and Kim, S. H. and Heja, S. and Readhead, B. and Dudley, J. T. and others},
  title   = {{Integrative approach to sporadic Alzheimer's disease: deficiency of TYROBP in cerebral Aβ amyloidosis mouse normalizes clinical phenotype and complement subnetwork molecular pathology without reducing Aβ burden}},
  journal = {Molecular psychiatry},
  year    = {2019},
  doi     = {10.1038/s41380-018-0255-6},
  pmid    = {30283032},
  url     = {https://doi.org/10.1038/s41380-018-0255-6}
}

@article{audrain2019-30283031,
  author  = {Audrain, M. and Haure-Mirande, J. V. and Wang, M. and Kim, S. H. and Fanutza, T. and Chakrabarty, P. and Fraser, P. and St George-Hyslop, P. H. and others},
  title   = {{Integrative approach to sporadic Alzheimer's disease: deficiency of TYROBP in a tauopathy mouse model reduces C1q and normalizes clinical phenotype while increasing spread and state of phosphorylation of tau}},
  journal = {Molecular psychiatry},
  year    = {2019},
  doi     = {10.1038/s41380-018-0258-3},
  pmid    = {30283031},
  url     = {https://doi.org/10.1038/s41380-018-0258-3}
}

@article{audrain2021-33314529,
  author  = {Audrain, M. and Haure-Mirande, J. V. and Mleczko, J. and Wang, M. and Griffin, J. K. and St George-Hyslop, P. H. and Fraser, P. and Zhang, B. and others},
  title   = {{Reactive or transgenic increase in microglial TYROBP reveals a TREM2-independent TYROBP-APOE link in wild-type and Alzheimer's-related mice}},
  journal = {Alzheimer's \& dementia : the journal of the Alzheimer's Association},
  year    = {2021},
  doi     = {10.1002/alz.12256},
  pmid    = {33314529},
  url     = {https://doi.org/10.1002/alz.12256}
}