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% Source: https://alzhub.org/research?author=Stavrides+P
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@article{lee2022-35654956,
  author  = {Lee, J. H. and Yang, D. S. and Goulbourne, C. N. and Im, E. and Stavrides, P. and Pensalfini, A. and Chan, H. and Bouchet-Marquis, C. and others},
  title   = {{Faulty autolysosome acidification in Alzheimer's disease mouse models induces autophagic build-up of Aβ in neurons, yielding senile plaques}},
  journal = {Nature neuroscience},
  year    = {2022},
  doi     = {10.1038/s41593-022-01084-8},
  pmid    = {35654956},
  url     = {https://doi.org/10.1038/s41593-022-01084-8}
}

@article{jiang2016-26923405,
  author  = {Jiang, Y. and Rigoglioso, A. and Peterhoff, C. M. and Pawlik, M. and Sato, Y. and Bleiwas, C. and Stavrides, P. and Smiley, J. F. and others},
  title   = {{Partial BACE1 reduction in a Down syndrome mouse model blocks Alzheimer-related endosomal anomalies and cholinergic neurodegeneration: role of APP-CTF}},
  journal = {Neurobiology of aging},
  year    = {2016},
  doi     = {10.1016/j.neurobiolaging.2015.11.013},
  pmid    = {26923405},
  url     = {https://doi.org/10.1016/j.neurobiolaging.2015.11.013}
}

@article{im2022-4ba4a0f3,
  author  = {Im, E. and Jiang, Y. and Stavrides, P. and Darji, S. and Erdjument-Bromage, H. and Neubert, T. A. and Choi, J. Y. and Wegiel, J. and others},
  title   = {{Lysosomal dysfunction in Down Syndrome and Alzheimer mouse models is caused by selective v-ATPase inhibition by Tyr 682 phosphorylated APP βCTF}},
  journal = {Science advances},
  year    = {2022},
  doi     = {10.1101/2022.06.02.494546},
  url     = {https://doi.org/10.1101/2022.06.02.494546}
}